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Table 1 Axon stalling rates in mutant and transgenic nrg embryos

From: The L1-type cell adhesion molecule Neuroglian is necessary for maintenance of sensory axon advance in the Drosophila embryo

 

Percentage of stalled axons (total n)*

 

Neuron

 

Genotype

lch5-1

lch5-5

dbd

All neurons combined (excluding dbd)

w 1118

0% (19)

0% (27)

0% (8)

-

nrg 14 /Y

48%† (21)

84%† (25)

0% (14)

70% (87)

nrg 17 /Y

-

79% (28)

-

71% (45)

nrg 14 /Y;P0163-GAL4/UAS-nrg 180

0% (22)

20%‡ (20)

-

-

nrg 14 /Y;UAS-nrg GPI ;P0163-GAL4

-

48%‡ (25)

-

-

P0163-GAL4/UAS-nrg 180

-

0% (31)

-

-

elav-GAL4/UAS-nrg 180

-

0% (30)

-

-

P0163-GAL4/UAS-nrg GPI

-

7% (27)

-

-

elav-GAL4/UAS-nrg GPI

-

6% (16)

-

-

sal-GAL4;UAS-nrg 180

-

0% (35)

-

 

btl-GAL4/UAS-nrg 180

-

0% (23)

-

-

  1. *Based on morphology of DiI injected neurons in mid-stage 16 to stage 17 embryos. Axons were judged to have stalled if they had failed to reach the CNS neuropile. See Figure 2a for locations of stalling points for different types of neurons. †These values are significantly different to each other (P < 0.01). ‡These values are significantly different to the nrg14/Y stalling rates (P < 0.01) and to each other (P = 0.05).
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Neural Development

ISSN: 1749-8104